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OUTSMARTING THE FEMALE FAT CELL: YOU CAN'T STARVE A FAT CELL
A fat cell awakens with a smile. "This may be my lucky day. It's Monday, she didn't eat breakfast, drank two diet sodas this morning, ate a salad with lemon juice for lunch, and now she's fantasizing about chocolate eclairs. She must have started a new dietspread the word to all 30 billion fat cells. Put up the barricades, recruit the extra forces... we must unite and thrive."
If fat cells could communicate (and sometimes it seems they can), this is a likely conversation they would have each time you start a new diet. No matter how hard you try to starve yourself, you cannot starve your fat cells. They won't let you. There is a built-in protective mechanism that ensures their survival.
To help you understand why it is impossible to starve a fat cell, let's take a giant step back in history to analyze the development of fat storage in the human body. I'm sure that you have heard of Darwin's "survival of the fittest" theory. The weak perished and the strong survived, passing on their survival traits and adapting to their environment over time. There are many animal examples of survival of the fittest: the giraffe's long neck to reach high foliage, the tree frog's skin color for camouflage, the skunk's scent to ward off enemies. When it comes to human adaptation, survival of the fittest may really be "survival of the fattest."
Thousands of years ago, periodic food shortages were a way of life. They were the result of famine, drought, and catastrophe. Many died from starvation, but some people lived. Who were more likely to live? Those people with more body fat and bigger fat cells. They had more calories stored to withstand the food shortage. So, the fatter people lived and passed along their traits to the next generation. As "the fatter the better" trait was passed along from generation to generation, it became stronger and more specializedand we became fatter beings.
Not only were there famines, but the famines were followed by feasting times. The body came to realize that the more efficiently it stored fat during feasting times, the fatter it would be and the more likely it would be to survive during a famine. The result of the famine/feast cycle on the development of our fat physiology is twofold:
- Those people with larger fat cells survived the famine and passed along their trait.
- Those people who were more efficient fat storers during feasting times survived the famine and passed along their trait.
We have inherited big, stubborn fat cells that love to store fat and hate to give it up.
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Womens health
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HOW FOODS CAN PREVENT BREAST CANCER: DECREASE ESTROGENS
Here's how to counter the ill effects of bad, recycled, chemical, and free estrogens.
Bad Estrogen
Even if you produce moderate to high amounts of estrogen, there is an emerging strategy to blunt its potency. You can actually channel your estrogen into good estrogen rather than bad estrogen by eating a diet high in cruciferous vegetables. Those include cauliflower, broccoli, and cabbage. Both exercise and low body fat also increase the production of good estrogen. Alcohol, polyunsaturated fats, and too much body fat all increase the production of bad estrogen.
Recycled Estrogen
When estrogen is transported from the bloodstream through the liver and into the bowel for disposal, it is assisted by large amounts of fiber in the bowel. That fiber binds to estrogen in the intestine so that the body cannot reabsorb it, ensuring that it is excreted with other waste products. However, when there is too little fiber in the diet, the estrogen remains free in the bowel and may be reabsorbed by the body into the bloodstream, raising the amount of estrogen in the bloodstream. A study at Tufts University showed that the more a woman's bowel movement weighed, the lower was her blood estrogen level. The assumption is that the increased weight of the bowel movement was due to the fiber.
Free Estrogen
The most effective way to decrease the amount of free estrogen in the blood is to build more of the carriers that bind estrogen in the blood and keep it from estrogen receptors. Lets look at the key strategies. The prime regulator of estrogen carriers is the hormone insulin, according to Banoo Parpia of the China-Cornell-Oxford Project. The lower you can drop your insulin, the more estrogen carriers your body manufactures. A low-fat diet also reduces the amount of free estrogen in healthy postmenopausal women. Soy also manufactures more carriers. A high-fiber diet helps to bind more free estrogen in your blood and keeps it at lower, safer levels. Many of these measures also decrease estrogen production, so you are cutting your cancer risk in at least two separate ways.
Chemical Estrogen
The most aggressive prevention includes avoiding animal and fish products with high fat contents that can pick up and concentrate large quantities of chemical estrogens and pesticides. The worst offenders and how to avoid them are found in the chapter "Step 8: Avoid Chemical Estrogens." Eating organic foods that have always been pesticide-free will help you to avoid contaminating breast fat. Washing all fruits and vegetables thoroughly will help remove pesticides. Since most women already have high stores of chemical estrogens in their breast fats there are two other strategies that have proved to be beneficial. First is breast-feeding, which flushes pesticides out of their storage site in breast fat. That does mean that your infant ingests milk with chemical estrogens, but pediatricians do not believe this is harmful. The most practical strategy of all is to consume large amounts of estrogen blockers such as soy, which block the effect of these chemicals at the estrogen receptors on breast cells.
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Womens health
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